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Alzheimer's disease is characterized in part by abnormal deposits in the brain of the peptide beta amyloid (Abeta), in the form of plaques. Recent work with a transgenic model of Abeta deposition has uncovered a remarkable capacity of antibodies to prevent or clear these deposits. These experiments offer the attractive possibility, albeit some way in the future, of using antibodies as a therapy in Alzheimer's disease.

Type

Journal article

Journal

Trends Pharmacol Sci

Publication Date

01/2001

Volume

22

Pages

2 - 3

Keywords

Alzheimer Disease, Amyloid beta-Peptides, Animals, Humans, Immunization, Mice, Mice, Transgenic, Peptide Fragments