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Tuberous sclerosis complex 1 (hamartin) is an effective endogenous neuroprotectant. Understanding the endogenous mechanism for neuroprotection mediated by hamartin may afford a novel approach to effective treatment of neurological diseases such as stroke, neurodegenerative diseases, and epilepsy, with possible applications to nonneurological conditions.

Original publication

DOI

10.1111/ijs.12130

Type

Journal article

Journal

Int J Stroke

Publication Date

08/2013

Volume

8

Pages

449 - 450

Keywords

acute stroke therapy, hamartin, ischemic stroke, neuroprotection, productive autophagy, treatment, Animals, Epilepsy, Humans, Neurodegenerative Diseases, Stroke, Tumor Suppressor Proteins