Cookies on this website

We use cookies to ensure that we give you the best experience on our website. If you click 'Accept all cookies' we'll assume that you are happy to receive all cookies and you won't see this message again. If you click 'Reject all non-essential cookies' only necessary cookies providing core functionality such as security, network management, and accessibility will be enabled. Click 'Find out more' for information on how to change your cookie settings.

Publications

Brain-derived and in vitro-seeded alpha-synuclein fibrils exhibit distinct biophysical profiles

Lee SS. et al, (2024)

Unfolded protein response markers Grp78 and eIF2alpha are upregulated with increasing alpha‐synuclein levels in Lewy body disease

Hrabos D. et al, (2024), Neuropathology and Applied Neurobiology, 50

Brain-derived and in vitro-seeded alpha-synuclein fibrils exhibit distinct biophysical profiles

Lee SS. et al, (2023)

Bioorthogonally Cross‐Linked Hyaluronan‐Laminin Hydrogels for 3D Neuronal Cell Culture and Biofabrication

Jury M. et al, (2022), Advanced Healthcare Materials, 2102097 - 2102097

Bioorthogonally Cross-Linked Hyaluronan-Laminin Hydrogels for 3D Neuronal Cell Culture and Biofabrication

Jury M. et al, (2021)

The Luminescent Conjugated Oligothiophene h-FTAA Attenuates the Toxicity of Different Aβ Species.

Sandin L. et al, (2021), Biochemistry

Inhibition of nSMase2 Reduces the Transfer of Oligomeric α-Synuclein Irrespective of Hypoxia

Sackmann V. et al, (2019), Frontiers in Molecular Neuroscience, 12

Lipid vesicles affect the aggregation of 4-hydroxy-2-nonenal-modified α-synuclein oligomers

Sardar Sinha M. et al, (2018), Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1864, 3060 - 3068

Alzheimer’s disease pathology propagation by exosomes containing toxic amyloid-beta oligomers

Sardar Sinha M. et al, (2018), Acta Neuropathologica, 136, 41 - 56

The Luminescent Oligothiophene p-FTAA Converts Toxic Aβ1–42Species into Nontoxic Amyloid Fibers with Altered Properties

Civitelli L. et al, (2016), Journal of Biological Chemistry, 291, 9233 - 9243

Protective properties of lysozyme on β-amyloid pathology: implications for Alzheimer disease

Helmfors L. et al, (2015), Neurobiology of Disease, 83, 122 - 133

Herpes simplex virus type 1 infection in neurons leads to production and nuclear localization of APP intracellular domain (AICD): implications for Alzheimer’s disease pathogenesis

Civitelli L. et al, (2015), Journal of NeuroVirology, 21, 480 - 490

Influenza virus replication in lung epithelial cells depends on redox-sensitive pathways activated by NOX4-derived ROS

Amatore D. et al, (2015), Cellular Microbiology, 17, 131 - 145

The basic N-terminal domain of TRF2 limits recombination endonuclease action at human telomeres

Saint-Léger A. et al, (2014), Cell Cycle, 13, 2469 - 2474

In vitro inhibition of herpes simplex virus type 1 replication by Mentha suaveolens essential oil and its main component piperitenone oxide

Civitelli L. et al, (2014), Phytomedicine, 21, 857 - 865

Infectious Agents and Neurodegeneration

De Chiara G. et al, (2012), Molecular Neurobiology, 46, 614 - 638

Sex Differences in the Response to Viral Infections: TLR8 and TLR9 Ligand Stimulation Induce Higher IL10 Production in Males

Torcia MG. et al, (2012), PLoS ONE, 7, e39853 - e39853

HSV-1 promotes Ca2+-mediated APP phosphorylation and Aβ accumulation in rat cortical neurons

Piacentini R. et al, (2011), Neurobiology of Aging, 32, 2323.e13 - 2323.e26

APP Processing Induced by Herpes Simplex Virus Type 1 (HSV-1) Yields Several APP Fragments in Human and Rat Neuronal Cells

De Chiara G. et al, (2010), PLoS ONE, 5, e13989 - e13989

The number of vertebrate repeats can be regulated at yeast telomeres by Rap1-independent mechanisms

Brevet V., (2003), The EMBO Journal, 22, 1697 - 1706

Include forthcoming?